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<?xml-stylesheet type="text/xsl" href="/sheet.xsl"?><rss version="2.0"><channel><title>The Evolution of Cybernetics</title><link>https://sifter.org/~simon/journal</link><description>A Journal by Simon Funk</description><lastBuildDate>Sun, 22 Mar 2026 11:46:03 GMT</lastBuildDate><generator>PyRSS2Gen-1.1.0</generator><docs>http://blogs.law.harvard.edu/tech/rss</docs><item><title>Covid Update: VFR</title><link>https://sifter.org/~simon/journal/20211119.h.html</link><description>Covid Update: VFR</description><guid isPermaLink="true">https://sifter.org/~simon/journal/20211119.h.html</guid><pubDate>Fri, 19 Nov 2021 00:00:00 GMT</pubDate><ns0:encoded xmlns:ns0="http://purl.org/rss/1.0/modules/content/">&lt;blockquote style="margin: 0 auto; max-width: 800px;" morss_own_score="2.569937369519833" morss_score="123.5986750816098"&gt;
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&lt;h2&gt;Friday, November 19, 2021&lt;/h2&gt;
&lt;h4&gt;&lt;em&gt;Covid Update: VFR&lt;/em&gt;&lt;/h4&gt;
&lt;/center&gt;
&lt;br&gt;
&lt;br&gt;
&lt;p&gt;
My Covid updates have stalled while I've been waiting for any
sort of all-cause mortality data to come out.  And waiting.  And
waiting.  The stark absence of such data is itself very revealing&lt;a href="https://sifter.org/~simon/journal/20211119.h.html#update2"&gt;[2]&lt;/a&gt;.
&lt;/p&gt;
&lt;p&gt;
But it turns out some slipped out late October, brought to my
attention yesterday by &lt;a href="https://alexberenson.substack.com/p/another-major-red-flag-about-covid"&gt;Alex
Berenson&lt;/a&gt;.
&lt;/p&gt;
&lt;p&gt;
I thought it was worth a more thorough analysis, so here it is:
&lt;/p&gt;
&lt;p&gt;
TLDR: If this data holds up, the vaccines are
killing more people than they are saving.  Curious?  Read on.
&lt;/p&gt;
&lt;p&gt;
The basic setup is this: 
&lt;/p&gt;
&lt;p&gt;
&lt;a href="https://papers.ssrn.com/sol3/papers.cfm?abstract_id=3949410"&gt;The study&lt;/a&gt;
in question looks at Scandinavian-thorough data for some four million Swedes,
and shows that vaccine efficacy starts out good but wanes over time, thus
serving the current narrative push for boosters.  It's a big study and, although
not yet published, appears to be fairly well done.  I expect it answers the
questions it asks reasonably well.
&lt;/p&gt;
&lt;p&gt;
But, of course, they're only allowed to ask the wrong questions, because that's
how sciencism works these days.
&lt;/p&gt;
&lt;p&gt;
So let's ask the right ones.  Bear with me as I zip through this -- the
reasons for it all will become evident below.
&lt;/p&gt;
&lt;p&gt;
On figure one (page 32 of the pdf at this time) they say:
&lt;/p&gt;
&lt;p&gt;
"From this cohort, 4,034,787 individuals were identified that had two shots of vaccine 
no later than August 5."
&lt;/p&gt;
&lt;p&gt;
From which:
&lt;/p&gt;
&lt;p&gt;
"Excluded: 3,939 Died within 14 days of [date of second dose]."
&lt;/p&gt;
&lt;p&gt;
Now, Alex does some back of the envelope calculations for what this
number ought to be, but rightly observes that timing matters since,
e.g., if these were all in the winter, the expected death rates would be higher
(over 4k per 2 weeks).  And he also relies on extrapolated data from
prior years which is hazardous when death rates and numbers are
steadily changing year to year as evidenced in my &lt;a href="https://sifter.org/~simon/journal/20210724.1.html"&gt;prior
covid update&lt;/a&gt;.
&lt;/p&gt;
&lt;p&gt;
So, let's not make any guesses.  Let's actually calculate how many
people should have died in that window.  Here's how we do it:
&lt;/p&gt;
&lt;p&gt;
First, we take the 
&lt;a href="https://ourworldindata.org/covid-vaccinations#fully-vaccinated-by-age"&gt;
number of Swedes fully vaccinated, by date and age group&lt;/a&gt;:
&lt;/p&gt;
&lt;p&gt;
&lt;a href="https://sifter.org/~simon/journal/ims/20211119/covid-fully-vaccinated-by-age.png"&gt;&lt;img src="https://sifter.org/~simon/journal/ims/20211119/covid-fully-vaccinated-by-age.png"&gt;&lt;/a&gt;&lt;/p&gt;
&lt;p&gt;
This results in about 4.5 million people who had their second
dose during the time window covered by the study, with an
average age of 59.1 and an average second-dose date of 2021-06-02.
The larger study group in the paper, selected from their ~4
million cohort, had an average age of 59.4 and
an average second-dose date of 2021-05-19.  So, our sample (the full
sample of all second-doses in Sweden) is very representative of the
study sample.
&lt;/p&gt;
&lt;p&gt;
Next, we combine that with the
&lt;a href="https://www.mortality.org/"&gt;&lt;em&gt;actual&lt;/em&gt; number of deaths&lt;/a&gt;
in Sweden during the study window:
&lt;/p&gt;
&lt;p&gt;
&lt;a href="https://sifter.org/~simon/journal/ims/20211119/stmf.png"&gt;&lt;img src="https://sifter.org/~simon/journal/ims/20211119/stmf.png"&gt;&lt;/a&gt;&lt;/p&gt;
&lt;p&gt;
This is not us extrapolating some expected number of deaths from
past years--this is the &lt;em&gt;actual&lt;/em&gt; number of all-cause deaths
in Sweden for each week of the study, by age group.  Also, we can (and
I did) further extrapolate the distribution of those deaths within
each age group using historical death distributions, which does not
change the number of deaths at all but lets us (to a reasonably good
approximation) distribute them more accurately among the mis-aligned
vaccination age groups.
&lt;/p&gt;
&lt;p&gt;
After adjusting for the 4/4.5 million ratio (since they did not
use quite &lt;em&gt;all&lt;/em&gt; of the fully vaxxed people like we are), the
net result is, to a very good approximation, the number of deaths
we should have seen in the study in the two weeks following the
second doses, &lt;em&gt;assuming there is no correlation with the vaccine
and all-cause mortality at all&lt;/em&gt;.  Indeed, if the vaccines lower
all-cause mortality in that window, as the CDC recently claimed,
then the expected number should be higher than the actual number
(3,939).
&lt;/p&gt;
&lt;p&gt;
Running the calcuations, we get an expected 2,640 deaths.
&lt;/p&gt;
&lt;p&gt;
That means there were 1,300 more deaths in the two weeks following
the second dose of the vaccine than there should be.  I say "should"
as opposed to merely "we might expect" because this is based on
&lt;em&gt;actual&lt;/em&gt; numbers of deaths (can I emphasize that too many
times?) and actual second-dose dates by fairly narrow age bands.  The
most significant assumption in this particular calculation
is that the vaccinated population is not a priori significantly more
at risk of death than average.
If you look at the study's demographics, the vaxxed group does in
fact appear to be a bit less healthy than their age-matched unvaxxed
counterparts, so this is a real confounder, however
the impact is not likely to be large here&lt;a href="https://sifter.org/~simon/journal/20211119.h.html#update1"&gt;[1]&lt;/a&gt; -- even if they represented
100% of all Sweden's fatalities (an absurd extreme) there is still
a significant deficit as Alex observed in his initial back-of-the-envelope
upper limit.  Furthermore, this is just for the second
dose, and only for the immediately following two weeks, which means
&lt;em&gt;we are under-counting by however many causal deaths happened after
the first dose or after two weeks&lt;/em&gt; (not unlikely for cardio issues).
Let's call that a wash even though I suspect the true number is even
worse.
&lt;/p&gt;
&lt;p&gt;
Now, we don't know anything about how those excess deaths were
distributed (over time or age), but we do know how many doses
likely caused them, so can calculate, for roughly the currently
fully-vaccinated population of Sweden as a whole (since that's
roughly our study cohort):
&lt;/p&gt;
&lt;p&gt;
VFR (vaccine fatality rate) = 0.03%
&lt;/p&gt;
&lt;p&gt;
Or about 1 extra death per 3,100 second doses, just in the first
two weeks after the dose.
&lt;/p&gt;
&lt;p&gt;
In the original Pfizer trial, this would equate to about 6
excess deaths (of 19k vaxxed).  But Pfizer used an atypically
healthy cohort with an average age of only 50.4, so we should expect less.
The &lt;a href="https://www.fda.gov/media/151733/download"&gt;actual difference was 5&lt;/a&gt;
(not 1 like their public study release claimed), and that's
assuming they didn't &lt;a href="https://www.bmj.com/content/375/bmj.n2635"&gt;bury
more&lt;/a&gt;.  (Yes, that's right -- all-cause mortality in the
Pfizer RCT was 24% higher in the vaxxed arm.  Nothing to see here,
move along.)
&lt;/p&gt;
&lt;p&gt;
If we assume Americans are biologically similar to Swedes, then,
based on the number of fully vaccinated,
that translates to about 63,000 Americans killed so far by the
second dose alone.  This is on the same order as a number of
other estimates (from VAERS, etc), which typically have put
the figure at about 120,000 conservatively for all doses and
latencies.
&lt;/p&gt;
&lt;p&gt;
So, yeah.
&lt;/p&gt;
&lt;p&gt;
But wait, there's more...
&lt;/p&gt;
&lt;p&gt;
The cited study also happens to show that vaccine efficacy
for symptomatic infection goes &lt;em&gt;negative&lt;/em&gt; by about
9 months--something we are also seeing in the UK data.
&lt;/p&gt;
&lt;p&gt;
&lt;a href="https://sifter.org/~simon/journal/ims/20211119/VE.png"&gt;&lt;img src="https://sifter.org/~simon/journal/ims/20211119/VE.png"&gt;&lt;/a&gt;&lt;/p&gt;
&lt;p&gt;
But
the efficacy against &lt;em&gt;hospitalization and death&lt;/em&gt; remains
positive throughout, which is the high point of these vaccines:
Even if they're doing &lt;a href="https://www.thelancet.com/journals/laninf/article/PIIS1473-3099(21)00648-4/fulltext"&gt;a crap job of preventing transmission&lt;/a&gt;,
they protect you, at least.  Right?
&lt;/p&gt;
&lt;p&gt;
Right?
&lt;/p&gt;
&lt;p&gt;
Turns out, NOPE.
&lt;/p&gt;
&lt;p&gt;
What they appear to do is kill you if you're weak or especially
sensitive to the spike protein, so that Covid can't kill you later.
Okay, that's wild extrapolation, but amiwrong?
&lt;/p&gt;
&lt;p&gt;
Take the following with a grain of salt because it
depends on how much covid is going around at the time, but:
&lt;/p&gt;
&lt;p&gt;
If we assume about 25% of hospitalizations lead to death (higher
numbers make the vax look better so I'm conservatively erring
on the high side), then even by six months after the vax, when
its effectiveness had significantly waned and so the bulk of
its benefit was over, fewer people in
the above study had been spared death-by-covid than were killed in the
first two weeks by the vaccine itself.
As a ballpark, for every 5,000 second doses they killed one person
on net.  That would be equivalent to about 40,000 people needlessly
dead in the US to date (depending on how similar the covid waves were).
&lt;/p&gt;
&lt;p&gt;
Bottom line, the vaccine efficacy for &lt;em&gt;all-cause death&lt;/em&gt;
appears to be negative across the board--you're less likely to die if you
don't take it.  (And there's every reason to believe the boosters
will be significantly worse.  Early signs support this.)
&lt;/p&gt;
&lt;p&gt;
So, yeah.
&lt;/p&gt;
&lt;p&gt;
How'bout them mandates?
&lt;/p&gt;
&lt;hr&gt;
&lt;p&gt;
&lt;a&gt;[1]&lt;/a&gt; Update 2021-11-20: If we shuffle the deaths between groups so that our
vaxxed population are twice as likely as the unvaxxed to die a priori,
the excess deaths drop from 1300 to 1170--only about 10% and with
negligible impact on the VFR.  So this is unlikely a substantial
bias.
&lt;/p&gt;
&lt;p&gt;
&lt;a&gt;[2]&lt;/a&gt; Update 2021-11-20: Re. the recent all cause mortality data from the UK,
see &lt;a href="https://www.youtube.com/watch?v=6umArFc-fdc"&gt;this overview&lt;/a&gt;.
(Note in particular the very final graph.)
Update 12/3: See also  &lt;a href="http://probabilityandlaw.blogspot.com/2021/12/possible-systematic-miscategorisation.html"&gt;Possible systematic miscategorisation of vaccine status raises concerns about claims of Covid-19 vaccination effectiveness&lt;/a&gt;
And &lt;a href="https://bartram.substack.com/p/the-importance-of-the-delay-between"&gt;The importance of the delay between infection and death in official statistics&lt;/a&gt;
&lt;/p&gt;
&lt;p&gt;
[3] Update 2021-11-22: Just found this study (October preprint) which tries
to estimate the VFR from correlations of excess mortality with vax uptake
across various countries, and comes up with 0.04%, which is very close
to my 0.03%:
&lt;a href="https://www.researchgate.net/publication/355581860_COVID_vaccination_and_age-stratified_all-cause_mortality_risk"&gt;COVID
vaccination and age-stratified all-cause mortality risk&lt;/a&gt;
&lt;/p&gt;
&lt;p&gt;
[4] Update 2022-07-14: &lt;a href="https://repec.its.waikato.ac.nz/wai/econwp/2211.pdf"&gt;The Rollout of COVID-19 Booster Vaccines is Associated
With Rising Excess Mortality in New Zealand&lt;/a&gt; estimates 0.016% VFR in New Zealand from the boosters.
&lt;/p&gt;
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&lt;/blockquote&gt;
</ns0:encoded></item><item><title>Derationalizing, Part 1: Question the Obvious</title><link>https://sifter.org/~simon/journal/20220125.1.h.html</link><description>Derationalizing, Part 1: Question the Obvious</description><guid isPermaLink="true">https://sifter.org/~simon/journal/20220125.1.h.html</guid><pubDate>Tue, 25 Jan 2022 00:00:00 GMT</pubDate><ns0:encoded xmlns:ns0="http://purl.org/rss/1.0/modules/content/">&lt;blockquote style="margin: 0 auto; max-width: 800px;" morss_own_score="2.951871657754011" morss_score="107.15187165775401"&gt;
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&lt;h2&gt;Tuesday, January 25, 2022&lt;/h2&gt;
&lt;h4&gt;&lt;em&gt;Derationalizing, Part 1: Question the Obvious&lt;/em&gt;&lt;/h4&gt;
&lt;/center&gt;
&lt;br&gt;
&lt;br&gt;
&lt;p&gt;
My reply to this question from an acquaintance:
&lt;/p&gt;
&lt;blockquote&gt;&lt;i&gt;
 As a young person, it's difficult to know about every topic that is
 discussed. What do you do as a young skeptic who holds few opinions? Do you
 become the Socrates in every discussion/argument while offering very few of
 your own opinions?
&lt;/i&gt;&lt;/blockquote&gt;
&lt;blockquote&gt;&lt;i&gt;
 How can you ever be ready to say "I believe x" if there's so much room to
 say "I don't know"? It's unlikely that you'll go through every source about
 any subject, so at what point should you be confident enough to form an
 opinion?
&lt;/i&gt;&lt;/blockquote&gt;
&lt;blockquote&gt;&lt;i&gt;
 What happens when you don't know what you don't know?
&lt;/i&gt;&lt;/blockquote&gt;
&lt;blockquote&gt;&lt;i&gt;
[..]
&lt;/i&gt;&lt;/blockquote&gt;
&lt;blockquote&gt;&lt;i&gt;
 I guess the question here is: which strategy are people most receptive to
 in an argument? Which will help you learn the most? Is there a combination
 of these that allows for maximum learning efficiency?
&lt;/i&gt;&lt;/blockquote&gt;
&lt;p&gt;
    Imo there is only one answer to any of these and it's the
answer to all of these, and you touched on it up top w/Socrates:
&lt;/p&gt;
&lt;p&gt;
    Make your only objective: to understand why people believe
what they do.  (And in particular, not to try to convince anyone
of anything before you've done that.)
&lt;/p&gt;
&lt;p&gt;
    Once you achieve that (which you may or may not ever with
any given person...), depending on what the answer is there
are usually one of three options:  1) You realize they are right,
2) You use what you've learned about why they believe what they
do to guide them to a new belief, 3) You realize the reasons they
believe things are entirely irrational and, chances are, you're
too honest to do what it would take to change their minds.
&lt;/p&gt;
&lt;p&gt;
    Generally speaking you will find that honest people will
self-correct before you get to the end.  That is, if you just
keep asking them about whatever bit isn't making sense to you,
and assuming their answers don't clear it up for you and change
your mind, they will crash into their own inconsistencies or
unsupported premises in their effort to answer your questions,
and will say "Oh, now that you mention it.... maybe I had that
part wrong."  So in practice, it's rare you ever get a chance
to exercise (2) because those people self-correct before you get
there.
&lt;/p&gt;
&lt;p&gt;
    There are like 10 such people on the planet.  I know 5
of them.
&lt;/p&gt;
&lt;p&gt;
    Most of the time you'll eventually end up at (3), which
is a hard end to reach because you'll rightly be very reluctant
to accept just how irrational people are, particularly the
academics who've been trained to present as rational.  Sadly (1)
will be more rare than you hope, too, for the same reason.
&lt;/p&gt;
&lt;p&gt;
    To give you a head start on understanding why (most)
people believe what they do, here's my distillation
after half a century of studying how people think:
&lt;/p&gt;
&lt;p&gt;
    1) No matter how many reasons most people have for what
they believe, the reality is belief is a feeling and all the
reasoning is post-rationalization.  Deep down, certain things
are "obvious" to them, and this &lt;em&gt;feeling&lt;/em&gt; of obviousness is
the stopping condition that keeps them from ruminating endlessly
into the most minute details or remote possibilities.  In other
words, the feeling of truth or obviousness is a key and necessary
feature of cognition, and without it we would be unable to look
up from our navel in time to eat.  But the problem is, very few
people recognize it for what it is--a subconsciously acquired
emotional response (as opposed to a necessarily sound
inference)--and they blindly heed its impulse, which is to stop
thinking about the "obvious" thing in question.
&lt;/p&gt;
&lt;p&gt;
    So, most people most of the time are not in a learning mode
at all, they are post-rationalizing what they already feel.
&lt;/p&gt;
&lt;p&gt;
    2) What most people feel is true is programmed into them
by their tribe--in this age via their chosen media and social
networks.  People in media know this, and use it (the caveat
on (3) above does not apply to them).  But it doesn't happen
the way you think: It's not about presenting evidence and making
rational arguments, because as I said that's not how people
actually think.  It's about adopting the desired "truth" as
a &lt;em&gt;presupposition&lt;/em&gt; and then portraying everything using that
light.  So "the terrorists stormed the church and held fifty
people captive for three days" brings our conscious attention
to the described action, but hidden from scrutiny is the choice
of "terrorists" vs. "revolutionaries", which carries with it a
great deal of emotional implications which directly program
the readers.  Even if the depictions don't seem super credible,
repetition will make them into reality, because every time
someone is exposed to a phrase or story like that, they
picture it in their mind's eye, and their cortex integrates
that experience as if it had been seen in the real world.
&lt;/p&gt;
&lt;p&gt;
    In other words, if you can get people to listen to your
stories for enough minutes of the day, they will integrate
the presuppositions (the background reality implied by your
stories) of those stories as their model of the real world.
Doesn't matter if they believe they are reading fact or
fiction.
&lt;/p&gt;
&lt;p&gt;
    And looping back, once those presuppositions are
programmed in, they define what is "obvious", and in turn
what people will no longer think about.
&lt;/p&gt;
&lt;p&gt;
    Again, most people who make it to positions of serious
power understand all of this very well, and the world is
run via these strings.  The people you'll encounter will
mostly be on the wrong end of those strings.
&lt;/p&gt;
&lt;p&gt;
    So, you can try asking people why they believe what they
do, but mostly their answers won't make sense (they will
form a logical chain, but that chain will be &lt;em&gt;just one of
many logical chains&lt;/em&gt; and not necessarily the most likely
one by any obvious metric), so there's a fair chance you
won't come to understand why they believe what they do by
asking them.  Instead, find out what media they consume,
and then go pay attention to how that media is crafted,
and you will probably have your answer.  Which is why, for
case (3), there's usually nothing you can do about it.
&lt;/p&gt;
&lt;p&gt;
    Just beware, once you pay attention to media at this
level enough to see what I'm talking about, you'll have
learned to perceive it in the same way you perceive a
chair when you walk into a room, without having to analyze
the legs and seat and logically deduce that there's a chair.
I.e., you won't be able to un-see it, and then you'll spend
the rest of your life living in Clown World where you can't
believe you're surrounded by people who blindly swallow/follow
this crap.
&lt;/p&gt;
&lt;p&gt;
&lt;a href="https://www.youtube.com/watch?v=g4XiKChyK7A"&gt;They Live&lt;/a&gt;
&lt;/p&gt;
&lt;p&gt;
    (But you won't be able to take these glasses off.)
&lt;/p&gt;
&lt;p&gt;
    All that said, to end on a more productive note and
to tie in what I said at the start:  Imo the best approach
to take is to just ask questions.  If you're actively
debating someone, focus on the most important one or very
few--people will throw a lot of chaff to distract you away
from their inconsistencies.  Just ignore the chaff (hard to
do!) and laser focus on the important question--whatever
most doesn't make sense to you about their stance.  I have
actually "won" a few debates this way, the sorts of debates
that are not normally ever won by either side, which is as
much a credit to them as to my approach because most will
break down into full cognitive dissonance first:
&lt;/p&gt;
&lt;blockquote&gt;&lt;i&gt;
   "I have learned that, for most people, if you inspire within them a
    conflict between what they feel is right and what they know is right,
	well, fireworks or waterworks are usually the result."   -Rik Ling
&lt;/i&gt;&lt;/blockquote&gt;
&lt;p&gt;
    Equally important is to be that person who is willing
to answer the important questions straight, and to admit
error when you can't.  You might just make it 11.
&lt;/p&gt;
&lt;p&gt;
    Oh, and also actionable:  To whatever degree all of the
above is accurate and representative of humans in general
including you and me, the primary control you have over what
you believe, and the quality and accuracy of what you believe,
is the media you expose yourself to.  This is a very tricky
decision to make well, because:
&lt;/p&gt;
&lt;blockquote&gt;&lt;i&gt;
    "One thing I've learned in the half-century of living
    and learning is that if society establishes an organized
    process, cheats will enter, and game the system."
        -- &lt;a href="https://jameslyonsweiler.com/2021/07/20/ghouling-targeted-study-retraction-is-biasing-scientific-literature/"&gt;James Lyons-Weiler&lt;/a&gt;
&lt;/i&gt;&lt;/blockquote&gt;
&lt;p&gt;
    The most rigorous and seemingly trustworthy institutions
are also the most insidiously corrupt.
&lt;/p&gt;
&lt;p&gt;
    Independent bloggers and journalists are probably your
best bet right now.  Pick a wide range of ones who think well,
and rely on their public position as aggregators to pull in
what matters.  Avoid the mainstream media like the NPC blue
pill it is.  (Once you start paying attention to slant, you
won't be able to stomach mainstream media anyway.  See Clown
World.)
&lt;/p&gt;
&lt;p&gt;
    Lastly... to make the implied explicit:  You can do a
lot to clean up your own belief system by simply being willing
to entertain any challenge to something you consider "obvious"
(at least long enough to make sure it's not a new angle you
haven't honestly considered before; and even if you have, it's
worth re-visiting if it comes from someone you consider sane).
I.e., there's probably no way to avoid the emotional aspect
of belief (and the subconscious absorption of presuppositions
from media), but you can largely work around/patch it simply
by understanding that that's how the (your) mind works.
&lt;/p&gt;
&lt;p&gt;
    Which is a really long-winded way of saying: don't be
over-confident about anything.
&lt;/p&gt;
&lt;br&gt;&lt;br&gt;&lt;center&gt;[&lt;a href="https://sifter.org/~simon/journal/20211119.h.html"&gt;&amp;lt;&amp;lt;&lt;/a&gt; | 
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&lt;/blockquote&gt;
</ns0:encoded></item><item><title>Derationalizing, Part 2: Check your Blind Spot</title><link>https://sifter.org/~simon/journal/20220125.2.h.html</link><description>Derationalizing, Part 2: Check your Blind Spot</description><guid isPermaLink="true">https://sifter.org/~simon/journal/20220125.2.h.html</guid><pubDate>Tue, 25 Jan 2022 00:00:00 GMT</pubDate><ns0:encoded xmlns:ns0="http://purl.org/rss/1.0/modules/content/">&lt;blockquote style="margin: 0 auto; max-width: 800px;" morss_own_score="2.9470588235294115" morss_score="44.94705882352941"&gt;
&lt;center&gt;
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&lt;h2&gt;Tuesday, January 25, 2022&lt;/h2&gt;
&lt;h4&gt;&lt;em&gt;Derationalizing, Part 2: Check your Blind Spot&lt;/em&gt;&lt;/h4&gt;
&lt;/center&gt;
&lt;br&gt;
&lt;br&gt;
&lt;p&gt;
    [Addendum to &lt;a href="https://sifter.org/~simon/journal/20220125.1.h.html"&gt;last time&lt;/a&gt;]
&lt;/p&gt;
&lt;p&gt;
    I glossed over one key point that helps make more sense of
the rest:
&lt;/p&gt;
&lt;p&gt;
    Recall my example of walking into a room and seeing (perceiving)
the chair in it.  How do we do that, and what does "perception" mean
exactly?  The answer (my opinion, but one I'm very confident in) is
that perception is the act of matching some inputs (the image hitting
your retinas) with your existing model of the world.  And by "model"
here I mean (something equivalent to) a generative model--i.e. something
that could "imagine" scenes from the world.
&lt;/p&gt;
&lt;p&gt;
    I.e., you don't walk into a room and see a chair in a strictly
bottom-up (concrete-to-abstract) way.  Rather, you walk into a room
expecting (being able to imagine, top-down) various things that might
be there, and expecting what they might look like, and in effect you
&lt;em&gt;cull&lt;/em&gt; this set of possible things down to the ones that match the
inputs you get.
&lt;/p&gt;
&lt;p&gt;
    Why this matters is:  People are &lt;em&gt;utterly incapable of
perceiving&lt;/em&gt; anything outside of their existing model.  (People can
update their models with time but this is slow and.. rare.)
&lt;/p&gt;
&lt;p&gt;
    It's a little harder to illustrate for something like a chair
(because we all have very detailed models of 3d objects in general)
although it's more applicable there than one might guess, but probably
the most accessible example is if you've ever tried to learn a new
language with a different phonetic base than you are used to:  You
will initially perceive the language in terms of your existing base,
and in general won't be able to properly group and differentiate some
of the sounds.  And by that I mean, someone native to the language
might be able to say two sounds that are clearly different to them,
and to you they will sound qualitatively the same.  With practice those
sounds begin to emerge as distinct, and eventually you will (effortlessly)
hear them as two different things (as your model slowly updates to include
them).  Now, someone with a more elaborate low-level model of phonetics
might be able to differentiate sounds from a new language immediately --
so it's not strictly true that you need experience with a particular thing
to be able to perceive it; rather, it just depends on where your particular
model has what level of detail.  For someone who grew up in a single
accent of a single language, there is no &lt;em&gt;need&lt;/em&gt; to have a finer-grained
model than the phonemes of that dialect, and so the brain generally won't
waste resources on that, and they will be the most "blind" to the nuances
of a novel language.
&lt;/p&gt;
&lt;p&gt;
    Anyway, where this becomes more relevant is with more abstract
things.  Consider all the nuance of life, social interaction, politics,
and so on, which are "invisible" to children, and which you learn to
see more and more of in better and better detail as you get older.  My
point here is to reflect on what you could see a few years back and
compare to what you can see now; there must be some examples of things
you just naturally perceive now which you remember being completely
unaware of a few years back?  If you can hone in on that contrast, then
realize the same applies between any two people -- there are ways that
the people around you are or will be like children compared to you in
terms of what you can easily see that is entirely invisible to them.
&lt;/p&gt;
&lt;p&gt;
    I'm being long-winded on this, but it's super important, because
we naturally tend to imagine that everybody sees the same world that
we do.  But the reality is they don't--things can be plain as day to
you (not even a judgment call, just what seems to be a direct
perception) and totally invisible to them, and vice versa.   If you
don't account for this, it's a lot harder to understand other people's
perspectives.  Once you do account for this, you can generally learn
the limits of other people's models--typically by tribe since people's
models are, as I mentioned, mostly absorbed from the presuppositions
of their social/media context.  And you can be on the lookout for
anywhere that others seem to be able to see something you don't, and
can proactively learn to see that thing.
&lt;/p&gt;
&lt;p&gt;
    The example I gave before was media slant.  I have now witnessed
multiple people transition from being blind to it to seeing it.  It
is the closest real-world analogy to the "red pill" Matrix moment.
In general, this only happens when they have been so personally
familiar with something that they were forced to see the media version
vs. the real version, especially as both evolved in real time.  This
serves as a training example, via the correlates between the nuances
of the media and the particular ways and places they are deviating
from or misrepresenting reality.  For some reason, some people go
through this but never develop that model.  That's called Gell-Mann
Amnesia.  But people whose brains haven't calcified, or whatever is
the issue, update their models and gain a new and permanent insight.
&lt;/p&gt;
&lt;p&gt;
    The same situation with media exists with academia and science,
unfortunately.  (I've been collecting the patterns of bias and
fraud for years; thinking of assembling them into a book some day.)
And the same principle applies here:  Most people simply can't see it,
because they've not spent the time looking closely enough at it to
build a model of how it works; and without that model, they simply
can't see it even when it's right in front of them.  Again, I've seen
people transition (red pill) and almost always it is due to some
direct personal experience where they knew something so well that
they could not escape seeing the contrast between what was true and
what was broadly and universally accepted, supported, and defended
to the death by "science".  (Until years later, when science finally
comes around, and declares that those who thought otherwise before
were right for the wrong reasons.)
&lt;/p&gt;
&lt;p&gt;
    So, bottom line, don't assume the person you're talking to
sees the same thing even if you're both looking right at the
same thing.  Study their tribe and how it models the world, and
you'll eventually figure out what they will and won't be able to
see; and once you know that, people make a lot more sense...
(And you still won't be able to teach them anything, alas.)
&lt;/p&gt;
&lt;br&gt;&lt;br&gt;&lt;center&gt;[&lt;a href="https://sifter.org/~simon/journal/20220125.1.h.html"&gt;&amp;lt;&amp;lt;&lt;/a&gt; | 
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&lt;/blockquote&gt;
</ns0:encoded></item><item><title>Covid Update: Vitamin D</title><link>https://sifter.org/~simon/journal/20220204.html</link><description>Covid Update: Vitamin D</description><guid isPermaLink="true">https://sifter.org/~simon/journal/20220204.html</guid><pubDate>Fri, 04 Feb 2022 00:00:00 GMT</pubDate><ns0:encoded xmlns:ns0="http://purl.org/rss/1.0/modules/content/">&lt;blockquote style="margin: 0 auto; max-width: 800px;" morss_own_score="2.4871794871794872" morss_score="37.72792022792023"&gt;
&lt;center&gt;
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&lt;h2&gt;Friday, February 04, 2022&lt;/h2&gt;
&lt;h4&gt;&lt;em&gt;Covid Update: Vitamin D&lt;/em&gt;&lt;/h4&gt;
&lt;/center&gt;
&lt;br&gt;
&lt;br&gt;
&lt;p&gt;
I've been mentioning vitamin D as a likely severe-Covid preventative
with &lt;a href="https://sifter.org/~simon/journal/20200904.html"&gt;increasing confidence&lt;/a&gt; since &lt;a href="https://sifter.org/~simon/journal/20200429.html"&gt;April, 2020&lt;/a&gt;.
Science Tribe representatives have consistently pushed back,
adamant that it's woo woo nonsense, that there is "no statistically significant effect" and
other such fine tools of motivated incompetence.  (Their supporting evidence
is always lame, but they refuse to peel that onion
any further than the dry skin on the outside which has
"vitamin D is not the cure you're looking for" sharpied on it.)
&lt;/p&gt;
&lt;p&gt;
Well here's the latest:
&lt;/p&gt;
&lt;p&gt;
&lt;a href="https://journals.plos.org/plosone/article?id=10.1371/journal.pone.0263069"&gt;Pre-infection 25-hydroxyvitamin D3 levels and association with severity of COVID-19 illness&lt;/a&gt;
&lt;/p&gt;
&lt;blockquote&gt;
&lt;i&gt;
"Patients with vitamin D deficiency (&amp;lt;20 ng/mL) were 14 times more likely to have severe or critical disease than patients with 25(OH)D &amp;gt;=40 ng/mL"
&lt;/i&gt;
(14x: 95% CI 4-51; p&amp;lt;0.001)
&lt;/blockquote&gt;
&lt;p&gt;
That alone would be not very interesting if very few people
had poor vitamin D status, but alas that's common enough not
only to account for many Covid deaths, but to account for
nearly all of them.  See particularly figure 2:
&lt;/p&gt;
&lt;p&gt;
&lt;a href="https://sifter.org/~simon/journal/ims/20220204/figure2.png"&gt;&lt;img src="https://sifter.org/~simon/journal/ims/20220204/figure2.png"&gt;&lt;/a&gt;&lt;/p&gt;
&lt;p&gt;
Remarkably similar to the plot I posted April 2020, and
once &lt;em&gt;again&lt;/em&gt; showing that there are essentially no critical (and
very few severe) Covid cases with strong (prior!) vitamin-D status.
(And recall that &lt;a href="https://www.sciencedirect.com/science/article/pii/S0960076020302764"&gt;this study&lt;/a&gt;
among others showed it to be a causal and effective intervention,
not merely an indicator of underlying poor health.)
&lt;/p&gt;&lt;p&gt;
It would seem that most Covid deaths were (and continue to be) easily
avoidable. Along with billions of pharma profits, but I digress.
&lt;/p&gt;
&lt;hr&gt;
&lt;p&gt;
[Update Feb 6: Since multiple people have replied that vit-D status is
just a proxy for BMI, I will highlight this excerpt from the above
study: "Finally, among patients with a BMI &amp;lt;30, 49.5% have a 25(OH)D &amp;lt;20 ng/mL compared to 63.6% among patients with a BMI &amp;gt;= 30+ (p = 0.07, 2-sided; p = 0.04, 1-sided). The intensity of the correlation between BMI and vitamin D deficiency does not indicate a strong correlation (Crammer's V = 0.117). As a result, 25(OH)D was not, and cannot be, used as a surrogate value for BMI."]
&lt;/p&gt;
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&lt;/blockquote&gt;
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